DEMENTIA AND PARKINSON'S DISEASE — PATHHOLOGICAL AND NEUROCHEMICAL CONSIDERATIONS
Identifieur interne : 002956 ( Main/Exploration ); précédent : 002955; suivant : 002957DEMENTIA AND PARKINSON'S DISEASE — PATHHOLOGICAL AND NEUROCHEMICAL CONSIDERATIONS
Auteurs : Niall P. Quinn [Royaume-Uni] ; Martin N. Rossor [Royaume-Uni] ; C David Marsden [Royaume-Uni]Source :
- British Medical Bulletin [ 0007-1420 ] ; 1986.
Abstract
The prevalence of dementia in Parkinson's disease is greater than that expected in an age-matched population. While the pathology of Parkinson's disease itself may cause cognitive deficits, this seems unlikely to be responsible for the excess prevalence of dementia. Coincidental Alzheimer's disease certainly occurs, but again does not appear to account for the excess of dementia. However, coincident subclinical degrees of Alzheimer change may combine with similar biochemical and neuronal loss due to Parkinson's disease to cause a deficit sufficient to lead to dementia. Such superimposition of the pathological and biochemical changes of the two diseases could account for a wide spectrum of clinical presentations of the “parkingsonian dement” or the “demented parkinsonian”.
Url:
DOI: 10.1093/oxfordjournals.bmb.a072104
Affiliations:
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Rossor</name></author><author><name sortKey="Marsden, C David" sort="Marsden, C David" uniqKey="Marsden C" first="C David" last="Marsden">C David Marsden</name></author></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno type="RBID">ISTEX:264F4868AE24999771F44EB2212521AD80FF0E26</idno><date when="1986" year="1986">1986</date><idno type="doi">10.1093/oxfordjournals.bmb.a072104</idno><idno type="url">https://api.istex.fr/document/264F4868AE24999771F44EB2212521AD80FF0E26/fulltext/pdf</idno><idno type="wicri:Area/Main/Corpus">002530</idno><idno type="wicri:Area/Main/Curation">002198</idno><idno type="wicri:Area/Main/Exploration">002956</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a">DEMENTIA AND PARKINSON'S DISEASE — PATHHOLOGICAL AND NEUROCHEMICAL CONSIDERATIONS</title><author><name sortKey="Quinn, Niall P" sort="Quinn, Niall P" uniqKey="Quinn N" first="Niall P" last="Quinn">Niall P. Quinn</name><affiliation wicri:level="3"><country>Royaume-Uni</country><placeName><settlement type="city">Londres</settlement><region type="country">Angleterre</region><region type="région" nuts="1">Grand Londres</region></placeName><wicri:orgArea>University Department of Neurology, Institute of Psychiatry and King's College School of Medicine</wicri:orgArea></affiliation></author><author><name sortKey="Rossor, Martin N" sort="Rossor, Martin N" uniqKey="Rossor M" first="Martin N" last="Rossor">Martin N. Rossor</name><affiliation wicri:level="3"><country>Royaume-Uni</country><placeName><settlement type="city">Londres</settlement><region type="country">Angleterre</region><region type="région" nuts="1">Grand Londres</region></placeName><wicri:orgArea>University Department of Neurology, Institute of Psychiatry and King's College School of Medicine</wicri:orgArea></affiliation></author><author><name sortKey="Marsden, C David" sort="Marsden, C David" uniqKey="Marsden C" first="C David" last="Marsden">C David Marsden</name><affiliation wicri:level="3"><country>Royaume-Uni</country><placeName><settlement type="city">Londres</settlement><region type="country">Angleterre</region><region type="région" nuts="1">Grand Londres</region></placeName><wicri:orgArea>University Department of Neurology, Institute of Psychiatry and King's College School of Medicine</wicri:orgArea></affiliation></author></analytic><monogr></monogr><series><title level="j">British Medical Bulletin</title><idno type="ISSN">0007-1420</idno><idno type="eISSN">1471-8391</idno><imprint><publisher>Oxford University Press</publisher><date type="published" when="1986">1986</date><biblScope unit="volume">42</biblScope><biblScope unit="issue">1</biblScope><biblScope unit="page" from="86">86</biblScope><biblScope unit="page" to="90">90</biblScope></imprint><idno type="ISSN">0007-1420</idno></series><idno type="istex">264F4868AE24999771F44EB2212521AD80FF0E26</idno><idno type="DOI">10.1093/oxfordjournals.bmb.a072104</idno><idno type="ArticleID">42.1.86</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0007-1420</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract">The prevalence of dementia in Parkinson's disease is greater than that expected in an age-matched population. While the pathology of Parkinson's disease itself may cause cognitive deficits, this seems unlikely to be responsible for the excess prevalence of dementia. Coincidental Alzheimer's disease certainly occurs, but again does not appear to account for the excess of dementia. However, coincident subclinical degrees of Alzheimer change may combine with similar biochemical and neuronal loss due to Parkinson's disease to cause a deficit sufficient to lead to dementia. Such superimposition of the pathological and biochemical changes of the two diseases could account for a wide spectrum of clinical presentations of the “parkingsonian dement” or the “demented parkinsonian”.</div></front></TEI><affiliations><list><country><li>Royaume-Uni</li></country><region><li>Angleterre</li><li>Grand Londres</li></region><settlement><li>Londres</li></settlement></list><tree><country name="Royaume-Uni"><region name="Angleterre"><name sortKey="Quinn, Niall P" sort="Quinn, Niall P" uniqKey="Quinn N" first="Niall P" last="Quinn">Niall P. Quinn</name></region><name sortKey="Marsden, C David" sort="Marsden, C David" uniqKey="Marsden C" first="C David" last="Marsden">C David Marsden</name><name sortKey="Rossor, Martin N" sort="Rossor, Martin N" uniqKey="Rossor M" first="Martin N" last="Rossor">Martin N. Rossor</name></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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